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Nancy S. Krieger, Ph.D.


About Me

Faculty Appointments

Research Associate Prof (Part-Time) - Department of Medicine, Nephrology (SMD)



PhD | Stanford University. Pharmacology. 1976

BA | Indiana University. Chemistry. 1972


Journal Articles

Increased bone density in mice lacking the proton receptor, OGR1

Krieger, N.S., Yao, Z., Kyker-Snowman, K., Kim, M.H., Boyce, B.F.,Bushinsky, D.A.

Kidney International. 2016; 89(3): 565-573.

Effect of potassium citrate on calcium phosphate stones in a model of hypercalciuria.

Krieger, N.S., Asplin,J.R., Frick, K.K., Granja, I., Culbertson, C.D., Ng, A., Grynpas, M.D., Bushinsky, D.A.

J. Amer. Soc. Nephrology. 2015; 26: 3001-3008.

Persistence of 1,25D-induced hypercalciuria in alendronate treated genetic hypercalciuric stone-forming rats fed a low calcium diet.

Frick, K.K., Asplin, J.R., Culbertson, C.D., Granja, I., Krieger, N.S., Bushinsky, D.A.

American Journal of Physiology. 2014; 306: F1081-1087.

1,25(OH)2D3 induces a mineralization defect and loss of bone mineral density in genetic hypercalciuric stone-forming rats.

Ng, A. H., Frick,K.K., Krieger, N.S., Asplin,J.R., Cohen-McFarlane,M., Culbertson,C.D., Kyker-Snowman,K., Grynpas,M.D., Bushinsky,D.A.

Calcified Tissue International. 2014; 94: 531-543.

1,25(OH)2D3-Enhanced hypercalciuria in genetic hypercalciuric stone-forming rats fed a low calcium diet

Frick, K.K.,John Asplin, J.R, Krieger, N.S., Culbertson,C.D., Asplin,D.A., Bushinsky, D.A.

Am. J. Physiol., Renal Physiol. 305(8):F1132-8, 2013. 2013; 305(8): F1132-1138.

Increased biological response to 1,25(OH)2D3 in genetic hypercalciuric stone-forming rats.

Frick, K.K., Asplin,J.R., Favus,M.J.,Culbertson,C.D. Krieger,N.S., Bushinsky, D.A.

Am. J. Physiol 304: F718-F726, 2013. 2013; 304: F718-726.

Metabolic acidosis increases fibroblast growth factor 23 in neonatal mouse bone

Krieger,N.S., Culbertson,C.D., Kyker-Snowman, K. and Bushinsky,D.A.

Am J Physiol Renal Physiol. 2012; 303(F431-436).

Pharmacologic inhibition of intracellular calcium release blocks acid-induced bone resorption.

Krieger,N.S. and Bushinsky, D.A.

Amer. J. Physiol. 300:F91-97, 2011. 2011; 300: F91-97.

Metabolic acidosis increases intracellular calcium in bone cells through activation of the proton receptor OGR1

Frick,K.K., Krieger,N.S., Nehrke,K. and Bushinsky,D.A.

J. Bone Min. Res. 2009; 24: 305-313.

Regulation of COX-2 mediates acid-induced bone calcium efflux in vitro.

Krieger,N.S., Frick,K.K., LaPlante,K. and Bushinsky,D.A.

J. Bone Min. Res. 22: 907-918, 2007. 2007; 22: 907-918.

Effect of cortisol on acid-induced bone resorption in vitro

Krieger, N.S., Frick, K.K., and Bushinsky,D.A.

J. Amer. Soc. Nephrology. 2002; 13: 2534-2539.


Nutrition and Bone Health (2014)

Chapter: Acid-Base Balance and Bone Health

Authors: Bushinsky, D.A. and Krieger, N.S.

Publisher: Humana Press 2014

Bone-Metabolic Functions and Modulators (2011)

Chapter: Renal Diseases and Bone, Emerging Therapeutics

Authors: Krieger, N.S. and Bushinsky,D.A

Publisher: Springer-Verlag 2011

Disorders of Bone and Mineral Metabolism, (2002)

Chapter: Integration of mineral metabolism in the adult

Authors: Bushinsky, D.A. and Krieger, N.S.

Publisher: Lippincott, Williams & Wilkins, New York 2002

Renal Osteodystrophy (1998)

Chapter: Bone formation, resorption and turnover

Authors: Krieger, N.S

Publisher: Lippincott-Raven Publishers, Philadelphia, PA 1998