Lab Members

Environmental lung diseases COPD and IPF pathogenesis and therapeutic targets: effects of lung cellular mitochondrial dysfunction, senescence, and molecular clock.

My research focuses on whether premature lung aging is a cause or a consequence of environmental exposures. I am interested in studying how lifestyle and environmental factors—such as smoking, vaping, air pollution, and occupational hazards—predispose individuals to pulmonary disorders including COPD, IPF, and lung cancer. Using in vivo and in vitro toxicology alongside single-cell and spatial technologies, I aim to uncover mechanisms of smoke-induced senescence and impaired tissue repair, identifying early biomarkers and therapeutic targets for COPD.