Division of Endocrinology, Diabetes & Metabolism Make Their Mark at Annual Endocrine Society Meeting
Wednesday, June 22, 2022
Faculty, fellows, and grad students from URMC recently attended the annual conference of the Endocrine Society in Atlanta, Georgia. The society is a global community of more than 18,000 physicians and scientists who are dedicated to hormone research. Our faculty and students presented their work through posters, talks, presentations, and workshops.
See a full list of presenters and their titles.
Of special note, Stephen Hammes, M.D., division chief of Endocrinology, served as the chair for the meeting, and on the final day officially became the president-elect of the Endocrine Society. Hammes was also a top influencer on social media. You can follow him on Twitter, @StephenHammes for news and research.
“I could not be more proud of the huge participation by our University of Rochester faculty and fellows at the annual Endocrine Society meeting,” said Hammes, “which was our first live meeting in 3 years. Meetings like this afford us the opportunity to learn from others around the globe, to show off the great work being done at Rochester, and to meet new friends and colleagues who will remain with us for the rest of our lives.”
Laura Calvi and Roman Eliseev Receive $2.3M Grant to Study Efferocytosis by Bone Marrow Stromal Cells
Thursday, June 2, 2022
CMSR and Wilmot Cancer Institute researchers, Drs. Laura Calvi and Roman Eliseev, received a 5-year multi-PI grant from the National Institute of Aging totaling $2.3M. The grant is focused on understanding how efferocytosis, i.e. phagocytosis of dead and dying cells, by bone marrow mesenchymal stromal/stem cells (MSC) affects bone homeostasis and bone aging. With help of co-Investigators, Drs. Hani Awad and Minsoo Kim and using molecular, cellular and mouse genetic tools, Drs. Calvi and Eliseev will test the hypothesis that phagocytosis/efferocytosis by MSCs is an important component of osteoimmunology; however when pathologically increased in aging, it causes MSC oxidative stress, mitochondrial dysfunction and senescence, thus contributing to bone loss.