Current Students
Research Interest:
Tau Post-Translational Modifications mediate cellular dysfunctions in Alzheimer’s Disease
Research Interest:
Exploring the neurophysiologic and neuropathologic basis of photophobia in a rare lysosomal storage disorder.
Research Interest:
Mitochondrial genetics and function as determinants of mesenchymal stem cell fate
Research Interest:
Inflammatory processes involved in the onset and development of Osteoarthritis
Research Interest:
The effect of mitochondrial dysfunction and oxidative stress on tau pathology.
Research Interest:
The role of desmosomal protein PKP2 on the alteration of nuclear envelope integrity and genomic organization in arrhythmogenic cardiomyopathy
Research Interest:
Mechanism in Dental Implant Failure and Biomedical Tissue Engineering Treatment in Peri-implantitis.
Research Interest:
Identifying the contributions of the mycobiome to skin in health and disease.
Bio:
https://quintanalab.bwh.harvard.edu/research/
Research Interest:
The effects of modulating Sigma-1 receptor signaling on pathogenic activities of astrocytes in Parkinson's Disease
Research Interest:
Transcription factor regulation of progenitor cells during development
Research Interest:
Elucidating the Epigenetic Mechanisms that Regulate stress and steady-state Erythropoiesis, with the overarching goal of ameliorating symptoms in hemoglobinopathies.
Research Interest:
Mechanisms underlying TANGO2 Deficiency Disorder in a C. elegans model.
Research Interest:
Investigating the underlying mechanisms associated with Pancreatic cancer-induced skeletal muscle wasting.
Research Interest:
Optimization of an in vitro model of Staphylococcal abscess communities to study bactericidal mechanisms
Research Interest:
Role of the cGAS-STING Pathway in Cytoplasmic Trafficking of Plasmids”
Research Interest:
Investigating the role of retinoic acid (RA) signaling in Thyroid Eye Disease (TED) and TED pathophysiology.
Research Interest:
Role of energy metabolism in regulating mesenchymal stem cell fate. Mitochondrial function in adipogenic versus osteogenic lineages.
